Public sample analysisNo sign-in requiredDemo inputDerived

See how Luneri turns one flagged biomarker into a clearer next step.

A focused demo case built from the real Analyze interpretation layer. LDL-C is the main out-of-range signal, with companion markers included for context.

Simulated sample

Simulated sample data for educational preview only. This is not a diagnosis, treatment plan, or replacement for clinical review.

How this is sourcedDemo input

This sample uses a fixed example panel while the interpretation layer still runs through the real analysis engine.

How this is sourcedDerived

The demo case uses fixed biomarker inputs, but the interpretation layer uses the same deterministic analysis engine and dossier-linked mapping as the main product.

Sample panel

Sample cardiometabolic review

5 markers entered

LDL Cholesterol

LDL

High

170

mg/dL

Above the current reference window.

Reference < 100

170 mg/dL

HDL Cholesterol

HDL

In range

52

mg/dL

Inside the current reference window.

Reference > 40

52 mg/dL

Triglycerides

TG

In range

118

mg/dL

Inside the current reference window.

Reference < 150

118 mg/dL

Fasting Glucose

Glucose

In range

92

mg/dL

Inside the current reference window.

Reference < 100

92 mg/dL

High-Sensitivity CRP

hs-CRP

High

1.4

mg/L

Above the current reference window.

Reference < 1

1.4 mg/L

Flagged biomarker

LDL needs intervention

LDL is 70% above 100 mg/dL at 170 mg/dL.

Top surfaced direction

Magnesium

A

LDL-C: small effect signal; meta-analysis or randomized trial evidence; dyslipidemia and cardiometabolic risk.

Why this surfaced

Chosen because indexed human evidence maps it to lowering LDL, and LDL-C: small effect signal; meta-analysis or randomized trial evidence; dyslipidemia and cardiometabolic risk.

Safety watch-out

LDL-C: small effect signal; adult dyslipidemia and hypertriglyceridemia contexts.

Report workflowReport previewCardiometabolicEducational sample

Report workflow preview

The report engine turns the same sample markers into ranked options, protocol hints, and caveats.

This preview is deterministic and evidence-linked. It is not a diagnosis, treatment plan, or replacement for clinical review.

Simulated sample

Report structure is real; the biomarker values on this page are demo data for workflow preview only.

Biomarker priorities

3 surfaced
HighHigh priority

LDL

LDL is high and is a primary Cardiometabolic marker.

HighHigh priority

hs-CRP

hs-CRP is high and is a primary Cardiometabolic marker.

In rangeMedium priority

HDL

HDL is normal and is a primary Cardiometabolic marker.

Evidence-ranked options

3 candidates
Rank 1Dossier-backedFit 317Protocol-ready

Berberine

Berberine matches hs-CRP with a A-grade decrease signal.

hs-CRPbeneficial
LDLbeneficial
Glucosecontextual
HDLcontextual
Rank 2Dossier-backedFit 315Protocol-ready

L Carnitine

L Carnitine matches hs-CRP with a A-grade decrease signal.

hs-CRPbeneficial
LDLbeneficial
HDLcontextual
TGcontextual
Rank 3Dossier-backedFit 314Protocol-ready

Taurine

Taurine matches hs-CRP with a A-grade decrease signal.

hs-CRPbeneficial
LDLbeneficial
Glucosecontextual
TGcontextual

Protocol seed hints

2
BerberineWith meals

glycemic control

500 mg berberine HCl

L CarnitineWith meals

General health supplementation — lipid optimization, anti-inflammatory support, metabolic health

1000–2000 mg/day

Safety and uncertainty

5
Medium

pgx-review

Low

Unmapped biomarker signal: 8-ohdg

Low

Unmapped biomarker signal: abeta-burden

Low

7 ranked interventions used registry fallback provenance.

Low

Unmapped biomarker signal: 8-ohdg

Full report sections

6
Biomarker summaryEvidence-ranked optionsProtocol considerationsSafety and cautionsFollow-up markersEvidence notes
Manual interpretationLipid PanelInflammationDerived

Clinical triage

1 marker needs attention now, with 1 additional marker worth addressing.

How this is sourcedDerived

This analysis is generated by deterministic biomarker logic using dossier-linked intervention mapping.

1need attention now
1marker needs intervention
3markers to monitor

Next steps

Clinical handoffs

Move from interpretation into research review, protocol construction, or follow-through.

Review Magnesium dossier

Start with the primary research brief tied to LDL.

Compare candidates

Pressure-test Magnesium vs Zinc before turning the result into a protocol.

Build seeded protocol

Seed the protocol builder with the strongest overlap and priority compounds.

Clinical follow-through

Recheck in 8-12 weeks

Follow through with ApoB, TG for a cleaner read on improvement.

Add more markers

Return to intake without clearing the current manual set and widen prioritization.

Immediate attentionLipid Panel70% above boundary

LDL

LDL Cholesterol

LDL is 70% above 100 mg/dL at 170 mg/dL.

Low-density lipoprotein cholesterol; primary atherogenic particle

Current value

Above range

170

mg/dL

70% above boundary

Reference < 100

170 mg/dL

Clinical meaning

Use Magnesium and Zinc as the first research-backed directions to lower LDL.

Reference < 100 mg/dL | Optimal < 70 mg/dL

Reference context

Reference < 100 mg/dL | Optimal < 70 mg/dL

Use Magnesium and Zinc as the first research-backed directions to lower LDL.

Recommended direction

Decision-oriented interventions prioritized by evidence strength, role, and current marker fit.

Magnesium

MetabolicPrimaryDerived

Glycinate and L-Threonate

A

Targets

LDL

Chosen because indexed human evidence maps it to lowering LDL, and LDL-C: small effect signal; meta-analysis or randomized trial evidence; dyslipidemia and cardiometabolic risk.

LDL-C: small effect signal; meta-analysis or randomized trial evidence; dyslipidemia and cardiometabolic risk.

How this is sourcedDerived

Some supporting biomarker mappings still fall back to the registry while dossier normalization is incomplete (65 unmapped signals).

Zinc

NutraceuticalAdjunctDerived

Structured dossier entry

A

Targets

LDL

Chosen because indexed human evidence maps it to lowering LDL, and LDL-C: Pooled WMD −0.20 to −0.38 mmol/L in T2D; inconsistent in non-diabetic populations; dose-response suggests stronger effect at higher doses; meta-analysis; T2D, dyslipidemia, adults.

LDL-C: Pooled WMD −0.20 to −0.38 mmol/L in T2D; inconsistent in non-diabetic populations; dose-response suggests stronger effect at higher doses; meta-analysis; T2D, dyslipidemia, adults.

How this is sourcedDerived

Dossier biomarker normalization still has 14 unmapped signals under review.

Zone 2 Cardio

ExerciseOptionalDerived

behavioral intervention

A

Targets

LDL

Chosen because indexed human evidence maps it to lowering LDL, and LDL-C: MD −0.1 to −0.3 mmol/L in overweight/obese; HIIT may produce larger LDL-C reductions than energy-matched MICT (Vella 2017: HIIT −0.66 vs MICT −0.03 mmol/L, p<0.05 in n=17); inconsistent with Wood 2019 MA showing no HIIT superiority for lipids — small sample signal only; meta-analysis; adults overweight obese, adults dyslipidemia.

LDL-C: MD −0.1 to −0.3 mmol/L in overweight/obese; HIIT may produce larger LDL-C reductions than energy-matched MICT (Vella 2017: HIIT −0.66 vs MICT −0.03 mmol/L, p<0.05 in n=17); inconsistent with Wood 2019 MA showing no HIIT superiority for lipids — small sample signal only; meta-analysis; adults overweight obese, adults dyslipidemia.

Safety watch-out

Omega 3 may push this marker in the wrong direction.

A
Derived

LDL-C: small effect signal; adult dyslipidemia and hypertriglyceridemia contexts.

How this is sourcedDerived

Dossier biomarker normalization still has 58 unmapped signals under review.

Clinical follow-through

Recheck in 8-12 weeks

Monitor

Improvement looks like LDL-C moving back toward range while ApoB and triglycerides trend in the same direction.

ApoBTG
Needs interventionInflammation40% above boundary

hs-CRP

High-Sensitivity CRP

hs-CRP is 40% above 1 mg/L at 1.4 mg/L.

Sensitive systemic inflammation marker; CVD risk predictor

Current value

Above range

1.4

mg/L

40% above boundary

Reference < 1

1.4 mg/L

Clinical meaning

Use Creatine and NAC as the first research-backed directions to lower hs-CRP.

Reference < 1 mg/L | Optimal < 0.5 mg/L

Reference context

Reference < 1 mg/L | Optimal < 0.5 mg/L

Use Creatine and NAC as the first research-backed directions to lower hs-CRP.

Recommended direction

Decision-oriented interventions prioritized by evidence strength, role, and current marker fit.

Creatine

RecoveryPrimaryDerived

Monohydrate

A

Targets

hs-CRP

Chosen because indexed human evidence maps it to lowering hs-CRP, and hs-CRP: small effect signal; meta analysis; healthy adults, athletes, older adults.

hs-CRP: small effect signal; meta analysis; healthy adults, athletes, older adults.

How this is sourcedDerived

Some supporting biomarker mappings still fall back to the registry while dossier normalization is incomplete (36 unmapped signals).

NAC

HepaticAdjunctDerived

N-Acetyl Cysteine

A

Targets

hs-CRP

Chosen because indexed human evidence maps it to lowering hs-CRP, and hs-CRP: moderate; randomized trial pooled; adult, renal failure, pcos.

hs-CRP: moderate; randomized trial pooled; adult, renal failure, pcos.

How this is sourcedDerived

Dossier biomarker normalization still has 68 unmapped signals under review.

HIIT Training

ExerciseOptionalDerived

behavioral intervention

A

Targets

hs-CRP

Chosen because indexed human evidence maps it to lowering hs-CRP, and hs-CRP: Pooled MD ~-0.5 to -1.5 mg/L in overweight/obese and metabolic disease; SMD ~-0.5; HIIT comparable to or modestly superior to MICT; obese children: MD ~-0.4 mg/L; multiple meta analyses; overweight obese, t2dm, metabolic syndrome.

hs-CRP: Pooled MD ~-0.5 to -1.5 mg/L in overweight/obese and metabolic disease; SMD ~-0.5; HIIT comparable to or modestly superior to MICT; obese children: MD ~-0.4 mg/L; multiple meta analyses; overweight obese, t2dm, metabolic syndrome.

Next step

Create a free account to move from a sample case into your own research workflow.

Start free, keep browsing dossiers, and unlock deeper analysis or protocol workflows when you are ready.

Simulated sample

The preview above uses demo values; your Analyze workspace starts with your own entered labs.